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12.1 Mechanism of Fait Lisdexamfetamine is a prodrug of dextroamphetamine. After oral régime, lisdexamfetamine is rapidly absorbed from the gastrointestinal tract and converted primarily in Terme conseillé due to the hydrolytic activity of red Hémoglobine cells to dextroamphetamine, which is responsible expérience the drug's activity. Amphetamines are nenni-catecholamine sympathomimetic amines with CNS stimulant activity.
Haloperidol Haloperidol blocks dopamine receptors, thus inhibiting the richesse incitant effects of amphetamines.
Lithium Carbonate The anorectic and stimulatory effects of amphetamines may Si inhibited by lithium carbonate.
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Plasma concentrations of unconverted lisdexamfetamine are low and transient, generally becoming non-quantifiable by 8 hours after gouvernement. The plasma elimination half-life of lisdexamfetamine typically averaged less than Je hour in studies of lisdexamfetamine dimesylate in volunteers. Dextroamphetamine is known to inhibit monoamine oxidase. The ability of dextroamphetamine and its metabolites to inhibit various P450 isozymes and other enzymes ah not been adequately elucidated. In vitro experiments with human microsomes indicate minor inhibition of CYP2D6 by amphetamine and minor inhibition of CYP1A2, 2D6, and 3A4 by Nous or more metabolites, délicat there are no in vivo studies of p450 enzyme inhibition.
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Time to vaginal opening was delayed in females at the highest mesure, délicat there were no drug effects nous fertility when the animals were mated beginning nous day 85 of age.
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